1. In the experiment group dissolution of myofibril is reduced.

2. Different degrees of myofibril damage were found.

3. And there were endoplasmatic reticulum enlargement and the myofibril contracture in the 4℃ group.

4. Ectodermal skin muscle cells in myofibril generally arranged along the axis of the body, the contraction of the body shorter.

5. Results: The content of protein, myofibril protein and myosinogen gradually descend, and nonprotein nitrogen(NPN) gradually increase along with the extend of fermented time.

6. LPS can damage rat myofibril and cause myocardial function decrease, PD has protective effect to these damages. ?

7. Meantime the property of the myofibril contraction under different biochemistry conditions had been tested.

8. CONCLUSION:Myocardial interstitial fibrosis and the reduction of myofibril elements are the major pathological basis for the impairment of cardiac function in patients with DCM.

9. Fig . 9 Myocardium cell of Procambarus clark ii . Showing striation of myofibril.

10. Results showed that the freshness decreases with increasing time being out of water and loosening tissue cells and myofibril fragmentation can be observed as well as the further particles formation.

11. All cells have secretory and absorptive functions besides 4ibblasts and granular cells in connective tissue, there are myofibril,(Sentencedict) collagenous fibril and unmyelinated fibers.

12. Injuries of capillaries promoted the formation of contraction band(CB) of myofibril and myocardial necrosis.

13. The inhibition of Myostatin expression can accelerate the myoblast proliferation and myofibril hypertrophy, which results in the skeletal muscle hyperplasia.