1. What therapies advocated for the treatment of vasovagal syncope?

2. Is nitric oxide involved in the vasovagal syncope?

3. Are there " risk factors " for vasovagal syncope?

4. With which scenarios is vasovagal syncope usually associated?

5. What premonitory signs and symptoms may exist for vasovagal syncope?

6. The major failure causes included puncture muff, severe vasovagal reactions, the absence of a distinct trigger zone and incomplete analgesia after the procaine test.

7. AbstractObjectiveTo determine whether midodrine hydrochloride therapy could prevent vasovagal syncope (VVS) in pediatric patients.

8. Vasovagal syncope ( VVS ) is a frequent clinic apsychia syndrome.

9. The major failure causes included technical difficulties, vasovagal reactions, the absence of a distinct trigger zone, and no analgesia induced by procaine test.

10. Postural hypotension and vasovagal syncope are the major benign causes of syncope.

11. Have more invasive therapies been suggested for the treatment of vasovagal syncope?

12. Are there any new initiatives into the triggers of vasovagal syncope?

13. Further investigations will need to the extent of nitric oxide's role in vasovagal syncope.

14. What therapies have been advocated for the treatment of vasovagal syncope?

15. Objeclive To study the hemodynamic and neurohormonal changes in patients with vasovagal syn- cope during the head-up tilt (HUT) table test the triggering mechanisms of syncope.

16. Conclusion This data shows that there is impaired baroreflex function in patients with vasovagal syncope, and this may be associated with reduced baroreceptor sensitivity and autonomic dysfunction.

17. What therapies appear most effective for the treatment of vasovagal syncope?